Give two pieces of evidence that support the monoamine hypothesis of depression and one piece of evidence that seems to conflict with the hypothesis
What will be an ideal response?
The actions and behavioral effects of reserpine (depletes monoamines, reduces manic symptoms, induces depression), MAOIs (allow cytoplasmic concentrations of monoamines to increase, reduce symptoms of depression), amphetamine (increases extraneuronal monoamine levels, enhances mood), and monoamine reuptake blockers (increase extraneuronal monoamine levels, reduce symptoms of depression) are superficially compatible with the hypothesis, but the fact that MAOIs and many antidepressants enhance monoamine levels within hours but exert their benefits in depressed individuals over several days or weeks is a problem for the hypothesis. Also, clinically effective antidepressants only elevate mood in depressed individuals. Also, treatments that acutely deplete the brain of NE or 5-HT in normal adults without a history of depression do not induce the core symptoms of depression.
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The abstinence-only sex-education program that was widely promoted in the United States in 1998 resulted in
a dramatic reduction in sexual activity by teenagers. a reduced incidence of sexual activity by teenagers. no significant impact on sexual activity by teenagers. increased sexual activity by teenagers.
The visual information from each eye is split at the __________________, with half of the nerve fibers from each eye projecting to each half of the brain
A) fovea B) optic chiasm C) lateral geniculate nucleus D) visual cortex